Vol. 1, Issue 3, Part A (2018)
Benign prostatic hyperplasia with respect to diet
Author(s): Daphal Supriya, Patil Anup and Gharge Varsha
Abstract: Benign Prostatic hyperplasia is not a cancerous, but it is a condition in a men in which prostatic gland enlarged. In which the gland pinches the urethra, so that the bladder becomes thicker, eventually bladder becomes weaker and lose the ability to empty completely. Usually it begins between the age of 30 -50 years. Testosterone is converted into Dihydrotestosterone in presence of 5-a reeducates inhibitor. Therapy with a 5 -a reeducates inhibitor markedly reduces the DHT content of prostate and in turn reduces the prostate volume. Generally low volume conc. of Testosterone is found in a Benign Prostatic Hyperplasia. Anatomically the median lobe is usually enlarged in BPH. The anterior lobe has little in the way of glandular tissue and is seldom enlarged carcinoma of the prostate typically occurring in posterior lobe. In Benign Prostatic Hyperplasia majority of growth occur in the TZ, In addition to this of classic areas, the peripheral zone of the prostate is also involved to a lesser extent. The clinical diagnosis of BPH is based on the history of Lower Urinary Tract Symptoms (LUTS), a digital rectal examination, and exclusion of other causes. The degree of LUTS does not necessarily correspond to the size of the prostate. The dynamic component of the BPH is controlled by a sympathetic nervous system i.e. Androgenic receptor blockers which are commonly used to alleviate these symptoms act by relaxing prostatic smooth muscle. To avoid such conditions some key nutrients will be involved such as Saw Palmetto, Pyeum, Zinc, Alanine, Glutamic acid /Glycine, Selenium, Starch. The explored possible diet and adaption of healthy life style will prevent the progression of BPH.
How to cite this article:
Daphal Supriya, Patil Anup and Gharge Varsha. Benign prostatic hyperplasia with respect to diet. International Journal of Advanced Community Medicine. 2018; 1(3): 29-37.